Myrrh for arthritis, and more

This looks interesting, especially with the impact of the gut bacteria info from the second link

http://biologicalexceptions.blogspot.com/2012/12/one-myrrh-aculous-christmas-gift.html

http://sciencehubb.co.uk/food-fight-bacterias-biological-warfare/

Looks like the proteins secreted by inflammation are the cause, and they just found a single one to try and inhibit.
Wish they would give a link for the chem layout..

http://m.medicalxpress.com/news/2013-05-discovery-fat-triggers-rheumatoid-arthritis.html

I hate the smell of myrrh but I use it with frankensense and black pepper and some others for my arthritis it works great. Real Lavender helps with the smell.

Bill Rahn wrote:I hate the smell of myrrh but I use it with frankensense and black pepper and some others for my arthritis it works great. Real Lavender helps with the smell.

Would you mind elaborating for me. I am interested on the degree of improvement in your arthritis and dosage regimes. Any helpful references, books and such would also be nice Thanks.

Morgan Morrigan wrote:This looks interesting, especially with the impact of the gut bacteria info from the second link


http://sciencehubb.co.uk/food-fight-bacterias-biological-warfare/

http://elife.elifesciences.org/content/2/e01202

Expansion of intestinal Prevotella copri correlates with enhanced susceptibility to arthritis

Animal models suggest a role for intestinal bacteria in supporting the systemic immune response required for joint inflammation. Here we performed 16S sequencing on 114 stool samples from rheumatoid arthritis patients and controls, and shotgun sequencing on a subset of 44 such samples. We identified the presence of Prevotella copri as strongly correlated with disease in new-onset untreated rheumatoid arthritis (NORA) patients. Increases in Prevotella abundance correlated with a reduction in Bacteroides and a loss of reportedly beneficial microbes in NORA subjects. We also identified unique Prevotella genes that correlated with disease. Further, colonization of mice revealed the ability of P. copri to dominate the intestinal microbiota and resulted in an increased sensitivity to chemically induced colitis. This work identifies a potential role for P. copri in the pathogenesis of RA.